
ACTH (4-10), human
CAS No. 4037-01-8
ACTH (4-10), human( Adrenocorticotropic Hormone (ACTH) (4-10), human )
Catalog No. M29938 CAS No. 4037-01-8
Adrenocorticotropic Hormone (ACTH) (4-10), human is a melanocortin 4 (MC4R) receptor agonist. Adrenocorticotropic Hormones (ACTH). ACTH stimulates the adrenal cortex and the secretion of glucocorticoids such as cortisol. Another name for ACTH is corticotropin.
Purity : >98% (HPLC)






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Biological Information
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Product NameACTH (4-10), human
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NoteResearch use only, not for human use.
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Brief DescriptionAdrenocorticotropic Hormone (ACTH) (4-10), human is a melanocortin 4 (MC4R) receptor agonist. Adrenocorticotropic Hormones (ACTH). ACTH stimulates the adrenal cortex and the secretion of glucocorticoids such as cortisol. Another name for ACTH is corticotropin.
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DescriptionAdrenocorticotropic Hormone (ACTH) (4-10), human is a melanocortin 4 (MC4R) receptor agonist. Adrenocorticotropic Hormones (ACTH). ACTH stimulates the adrenal cortex and the secretion of glucocorticoids such as cortisol. Another name for ACTH is corticotropin.(In Vitro):Adrenocorticotropic Hormone (ACTH) (4-10), human (ACTH (4-10)) is the core sequence of all melanocortins and binds selectively to MC4-R. In humans, ACTH (4-10) enters the cerebral fluid compartment after intranasal application and leads to a decrease in total body fat after 6 weeks of continuous treatment.
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In Vitro——
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In Vivo——
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SynonymsAdrenocorticotropic Hormone (ACTH) (4-10), human
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PathwayGPCR/G Protein
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TargetMelanocortin Receptor
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Recptor——
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Research Area——
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Indication——
Chemical Information
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CAS Number4037-01-8
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Formula Weight962.09
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Molecular FormulaC44H59N13O10S
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Purity>98% (HPLC)
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SolubilityIn Vitro:?DMSO : 25 mg/mL (25.99 mM)
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SMILES——
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Chemical NameSequence:Met-Glu-His-Phe-Arg-Trp-Gly
Shipping & Storage Information
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Storage(-20℃)
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ShippingWith Ice Pack
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Stability≥ 2 years
Reference
Wellh?ner P, et al. Intranasal application of the melanocortin 4 receptor agonist MSH/ACTH(4-10) in humans causes lipolysis in white adipose tissue. Int J Obes (Lond). 2012 May;36(5):703-8.
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